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The lymph nodes of the mediastinum, the spinal column are affected. Severe manifestations of Gabapentin lining of the aortic vessels. Aortitis accompanied by Takayasu's disease. It is often a hereditary disease, proceeds slowly. The main complication is arterial hypertension.

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In some patients, the pain is localized under the shoulder blades, in the knee joints, sometimes accompanied by an increase in body temperature, which, if misdiagnosed, can be masked as diseases of the musculoskeletal system.

To clarify the diagnosis, laboratory and instrumental methods are important: aortography; biochemical and general analysis of neurontin pills, urine; doppler scanning; serological examination; ultrasound procedure; electrocardiogram. In each case, certain methods are selected to establish and confirm the diagnosis. In the general blood test, some patients have an accelerated ESR and a large number of leukocytes, and in the biochemical - C-reactive protein, markers of the inflammatory process.

Treatment of gabapentin is first of all directed to the elimination of the provoking factor.

Recommended anti-inflammatory drugs and analgesics, immunostimulants, immunosuppressants, various antibiotics, mostly broad-spectrum. In allergic aortitis, glucocorticoid drugs are needed, as well as immunosuppressants. With such serious complications as the development of an aneurysm, treatment is effective only in the case of gabapentin intervention with further prosthetics of the affected area after resection. If there is aortic stenosis, then balloon dilatation, bypass, or stenting is possible. Possible complications, outcome.

The development and nature of complications depend on the form of the disease, neglect, age of the patient, and the treatment itself.

Inadequate treatment, as well as a neglected condition, can lead to the following consequences: myocardial infarction; stenosis of the coronary arteries; acute heart failure; the occurrence of blood clots in the arteries; violation of the blood flow of the brain; sudden death. The risk group for the development of complications primarily includes people who abuse smoking and alcohol, people with uncontrolled pressure, atherosclerosis, and a defect in collagen development.

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To prevent the occurrence of serious complications, first of all, therapy and timeliness in the treatment of primary diseases, the detection of tuberculosis, checking the body for viruses or other pathogens, and periodic visits to the doctor are important. Was the page helpful? The causes of aplasia are external teratogenic factors (physical, chemical, biological) that affect the embryo directly or indirectly through the mother's body. The same factors, acting on the gametes of parents (or more distant ancestors), can cause chromosomal diseases with aplasia of neurontin pills or another organ (tissue) or a hereditarily caused congenital malformation with aplasia.

Aplasia can be observed in any human organ (brain, spinal cord, heart, lungs, urinary-genital organs, and others). Aplasia of the heart (acardius, acardius) is observed only with twin congenital malformations. Aplasia of one of the paired organs usually causes vicarious hypertrophy of the other. Aplasia of one endocrine gland can cause hypo- or hypertrophy of other glands. There may be aplasia of only part of an organ, for example, the olfactory brain (arynencephaly), the corpus callosum, and so on.

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Focal tissue aplasia can be observed in the skin, most often on the scalp, where a defect is found at birth, usually not exceeding 5 cm in diameter. Skin aplasia can be combined with aplasia of deeper tissue, for example, in various forms of dysraphia (non-closure of embryonic fissures).